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Cyclosporine adversely affects baroreflexes via inhibition of testosterone modulation of cardiac vagal control.
TLDR
It is suggested that cyclosporine attenuates baroreflex responsiveness via, at least partly, inhibition of the testosterone-induced facilitation of cardiomotor vagal control.
Testosterone Facilitates the Baroreceptor Control of Reflex Bradycardia: Role of Cardiac Sympathetic and Parasympathetic Components
TLDR
Data provide the first experimental evidence of a favorable role for testosterone in baroreceptor control of reflex bradycardia, and baroreflex modulation by testosterone appears to be autonomically mediated and involves an enhancement of cardiomotor vagal activity.
Vasopressin and oxytocin in the neural control of the circulation.
TLDR
It is found that baroreceptor regulation of neuroendocrine and autonomic functions can be dissociated and a complex role of vasopressin (and possibly oxytocin) in the central neural regulation of the heart and circulation is suggested.
Contrasting effects of vasopressin on baroreflex inhibition of lumbar sympathetic nerve activity.
TLDR
Results indicate that intravenous vasopressin given for a period of 6 min facilitates the reflex inhibition of LSNA mediated through arterial baroreceptors in rabbits, but not in rats, and this facilitation appears to involve a central mechanism.
Regional and Endothelial Differences in Cyclosporine Attenuation of Adenosine Receptor-Mediated Vasorelaxations
TLDR
It is demonstrated that cyclosporine attenuates endothelium-dependent vasorelaxations elicited via activation of adenosine receptors and highlighted the interesting possibility that the relative contribution of the endothelial relaxing factors to cyclospora-NECA interaction is largely region dependent.
Cyclosporine attenuates the autonomic modulation of reflex chronotropic responses in conscious rats.
TLDR
This study provides the first pharmacological evidence that CyA attenuates reflex chronotropic responses via impairment of the autonomic modulation of the baroreceptor neural pathways.
Inhibition of canine adrenergic transmission by an analog of dopamine: GJH-166.
TLDR
The suggestion has been made that GJH-166 impairs sympathetic transmission by acting on certain inhibitory dopaminergic receptors located on the adrenergic nerve terminals.
Possible Ameliorative Effect of Ivabradine on the Autonomic and Left Ventricular Dysfunction Induced by Doxorubicin in Male Rats
TLDR
In conclusion, ivabradine ameliorated the LV dysfunction induced by doxorubicin, increased the overall HRV and restored the autonomic balance by reducing the sympathetic over activation, and may have a possible therapeutic potential against doxorbradine-induced heart failure.
Central mechanisms in DOC-salt hypertensive rats.
TLDR
The data suggested that selective central amplification of sympathetic vasomotor responses to centrally injected AII stimuli may play a role in the hypertension after 3 weeks of DOC-salt treatment in rats.
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