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A. de Bartolomeis1
I Marabese1
1A. de Bartolomeis
1I Marabese
1V. Di Marzo
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Enhanced supraspinal glutamate levels following nerve injury are associated with pathophysiological mechanisms responsible for neuropathic pain. Chronic pain can interfere with specific brain areas involved in glutamate-dependent neuropsychological processes, such as cognition, memory, and decision-making. The medial prefrontal cortex (mPFC) is thought to(More)
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