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Classification of Proliferative Pulmonary Lesions of the Mouse
Rapid advances in generating new mouse genetic models for lung neoplasia provide a continuous challenge for pathologists and investigators. Frequently, phenotypes of new models either have noExpand
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Induction of carcinogenesis by concurrent inactivation of p53 and Rb1 in the mouse ovarian surface epithelium.
Alterations in p53 and Rb pathways are observed frequently in epithelial ovarian cancer (EOC).However, their roles in EOC initiation remain uncertain. Using a single intrabursal administration ofExpand
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Mammary carcinoma suppression by cellular retinoic acid binding protein-II.
Retinoic acid (RA) modulates cell proliferation, differentiation, and apoptosis, and is used in chemotherapy and chemoprevention in several human cancers. RA exerts its pleiotropic activities byExpand
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Suppression of melanotroph carcinogenesis leads to accelerated progression of pituitary anterior lobe tumors and medullary thyroid carcinomas in Rb+/- mice.
Mice with a single copy of the retinoblastoma gene (Rb(+/-)) develop a syndrome of multiple neuroendocrine neoplasia. They usually succumb to fast-growing, Rb-deficient melanotroph tumors of theExpand
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Advances in Brief Induction of Carcinogenesis by Concurrent Inactivation of p 53 and Rb 1 in the Mouse Ovarian Surface Epithelium 1
Alterations in p53 and Rb pathways are observed frequently in epithelial ovarian cancer (EOC). However, their roles in EOC initiation remain uncertain. Using a single intrabursal administration ofExpand
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Pathology of Mouse Models of Human Lung Cancer.
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Binding Protein-II Mammary Carcinoma Suppression by Cellular Retinoic Acid Updated Version
Retinoic acid (RA) modulates cell proliferation, differentiation, and apoptosis, and is used in chemotherapy and chemoprevention in several human cancers. RA exerts its pleiotropic activities byExpand