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Extensive Cortical Rewiring after Brain Injury
TLDR
The results suggest that M1 injury results in axonal sprouting near the ischemic injury and the establishment of novel connections within a distant target, and support the hypothesis that, after a cortical injury, such as occurs after stroke, cortical areas distant from the injury undergo major neuroanatomical reorganization. Expand
Acute and chronic estrogen supplementation decreases uterine sympathetic innervation in ovariectomized adult virgin rats.
TLDR
The results indicate that sympathetic nerves are lost from uterine smooth muscle after estradiol treatment in a manner similar to that seen in the intact animal during estrus and pregnancy, which suggests that the rise inEstradiol prior to estrus is sufficient to deplete uterine sympathetic innervation. Expand
Post-infarct cortical plasticity and behavioral recovery using concurrent cortical stimulation and rehabilitative training: A feasibility study in primates
TLDR
Results support the feasibility of using a therapy approach combining peri-infarct electrical stimulation with rehabilitative training to alleviate chronic motor deficits and promote recovery from cortical ischemic injury. Expand
Variations in uterine innervation during the estrous cycle in rat.
TLDR
The estrous cycle in rat is accompanied by structural remodeling of sympathetic nerves by way of retraction or degeneration of terminal fibers during estrus, apparently preceded by depletion of catecholamine-synthesizing enzyme. Expand
Effects of small ischemic lesions in the primary motor cortex on neurophysiological organization in ventral premotor cortex.
TLDR
The results suggest that after a lesion in the M1 DFL, the induction of representational plasticity in PMv, as evaluated using intracortical microstimulation, is related more to the size of the lesion than to the disruption of its intrACortical connections. Expand
Neuronal HIF-1α Protein and VEGFR-2 Immunoreactivity in Functionally Related Motor Areas following a Focal M1 Infarct
TLDR
While remote areas undergo a molecular response to the infarct, it is hypothesized that there is a delay in the initiation of the response, which ultimately may increase the ‘window of opportunity’ for neuroprotective interventions in the intact cortex. Expand
Variations in uterine innervation during the estrous cycle in rat
TLDR
The estrous cycle in rat is accompanied by structural remodeling of sympathetic nerves by way of retraction or degeneration of terminal fibers during estrus, apparently preceded by depletion of catecholamine‐synthesizing enzyme. Expand
Sympathetic hyperinnervation of the uterus in the estrogen receptor alpha knock-out mouse.
TLDR
It is concluded that estrogen acting through the estrogen receptor alpha determines the number of sympathetic nerve terminal branches within uterine smooth muscle target, and in mice with low circulating estrogen, or high estrogen but lacking the functional estrogen receptoralpha, the uterus contains abundant sympathetic nerves. Expand
A Single Injection of d-Amphetamine Facilitates Improvements in Motor Training Following a Focal Cortical Infarct in Squirrel Monkeys
TLDR
D-AMPH is a potent modulator of behavioral recovery following an ischemic infarct in nonhuman primates as well as a pharmacological treatment to supplement rehabilitative therapy following stroke. Expand
Sympathetic hyperinnervation of the uterus in the estrogen receptor α knock-out mouse
TLDR
It is concluded that estrogen acting through the estrogen receptor α determines the number of sympathetic nerve terminal branches within uterine smooth muscle target, and in mice with low circulating estrogen, or high estrogen but lacking the functional estrogen receptorα, the uterus contains abundant sympathetic nerves. Expand
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