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Stimulation of 3T3 cells induces transcription of the c-fos proto-oncogene
Transcription of the c-fos proto-oncogene is greatly increased within minutes of administering purified growth factors to quiescent 3T3 cells. This stimulation is the most rapid transcriptional…
Association of Myn, the murine homolog of Max, with c-Myc stimulates methylation-sensitive DNA binding and ras cotransformation
Novel Anchorage of GluR2/3 to the Postsynaptic Density by the AMPA Receptor–Binding Protein ABP
c‐Myc represses transcription in vivo by a novel mechanism dependent on the initiator element and Myc box II.
It is shown that Myc‐dependent transactivation is insufficient for Ras cooperation and the novel transcription repression function is implicated in Ras cooperation as well as the suppression of Inr‐dependent genes.
PICK1 Targets Activated Protein Kinase Cα to AMPA Receptor Clusters in Spines of Hippocampal Neurons and Reduces Surface Levels of the AMPA-Type Glutamate Receptor Subunit 2
- J. L. Pérez, L. Khatri, Craig K. Chang, S. Srivastava, P. Osten, E. Ziff
- Biology, ChemistryThe Journal of Neuroscience
- 1 August 2001
It is indicated that PICK1 functions as a targeting and transport protein that directs the activated form of PKCα to GluR2 in spines, leading to the activity-dependent release of GLUR2 from synaptic anchor proteins and the P Pick1-dependent transport of Glu R2 from the synaptic membrane.
PICK1 Interacts with ABP/GRIP to Regulate AMPA Receptor Trafficking
RNA Editing at Arg607 Controls AMPA Receptor Exit from the Endoplasmic Reticulum
Recognition by Max of its cognate DNA through a dimeric b/HLH/Z domain
The three-dimensional structure of the basic/helix–loop–helix/leucine zipper domain of the transcription factor Max complexed with DNA has been determined by X-ray crystallography at 2.9 Å…
Mutagenesis Reveals a Role for ABP/GRIP Binding to GluR2 in Synaptic Surface Accumulation of the AMPA Receptor