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Sexual Differentiation
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Compensations after lesions of central dopaminergic neurons: some clinical and basic implications
View on Elsevier
Oxytocin produces natriuresis in rats at physiological plasma concentrations.
TLDR
The hypothesis that peripherally and centrally secreted OT act in concert in rats to produce a negative sodium balance by stimulating sodium excretion while inhibiting sodium ingestion is supported.
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Neurochemical compensation after nigrostriatal bundle injury in an animal model of preclinical parkinsonism.
TLDR
In these studies, damage to the nigrostriatal bundle of the rat was accompanied by apparent increases in the synthesis and release of dopamine from those dopaminergic terminals that remained, and both the activity of the rate-limiting biosynthetic enzyme and the content of the principal DA metabolite were increased in striatum relative to DA levels.
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Cholecystokinin activates catecholaminergic neurons in the caudal medulla that innervate the paraventricular nucleus of the hypothalamus in rats
TLDR
Results indicate that PVN‐projecting catecholaminergic neurons within the caudal medulla are activated by periph eral administration of CCK, further implicating these ascending catechlaminergic path ways in the neuroendocrine, physiological, and behavioral effects produced by gastric vagal stimulation.
View on Wiley
Medullary c-Fos activation in rats after ingestion of a satiating meal.
TLDR
Findings indicate that anatomically distinct subsets of hindbrain catecholaminergic neurons are activated in rats after food ingestion and that activation of these cells is quantitatively related to the magnitude of feeding-induced gastric distension.
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Cholecystokinin and gastric distension activate oxytocinergic cells in rat hypothalamus.
TLDR
Observations that the effects of CCK-8 on OXY release were attenuated by gastric vagotomy clearly demonstrate the existence of a sensitive neural link between the stomach and the neurohypophysis in the rat.
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Distribution and neurochemical phenotypes of caudal medullary neurons activated to express cFos following peripheral administration of cholecystokinin
TLDR
By characterizing the subnuclear distribution and chemical phenotypes of neurons activated by exogenous cholecystokinin, these data contribute to elucidation of the neural circuits mediating the behavioral, physiological, and neuroendocrine effects produced by this peptide.
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Brain oxytocin receptor antagonism blunts the effects of anorexigenic treatments in rats: evidence for central oxytocin inhibition of food intake.
TLDR
The hypothesis that pituitary secretion of OT after anorexigenic treatments in rats is associated with coactivation of centrally projecting brain OT pathways, some of which are causally related to the induced inhibition of food intake, is supported.
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Central oxytocin inhibition of angiotensin-induced salt appetite in rats.
TLDR
It is demonstrated that central administration of small doses of ANG II activates both magnocellular and parvocellular OT neurons in rats and indicates that some of the activated central OT pathway(s) may mediate an inhibitory effect that limits the salt ingestion induced by this treatment.
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