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Evaluation of mitochondrial respiratory chain activity in muscle healing by low-level laser therapy.
TLDR
The results suggest that the treatment with low-level laser may induce an increase in ATP synthesis, and that this may accelerate the muscle healing process.
Preconditioning Prevents the Inhibition of Na+,K+-ATPase Activity after Brain Ischemia
TLDR
Interestingly, ischemic preconditioning prevented the inhibitory effect of ischemia/reperfusion on Na+,K+-ATPase activity observed either after a single insult of 2 min or 10 min, suggesting that the maintenance of Na+K-ATpase activity afforded by preconditionsing be related to cellular neuroprotection.
Mitochondrial Dysfunction and Psychiatric Disorders
TLDR
Energy impairment as a mechanism underlying the pathophysiology of some psychiatric disorders, like bipolar disorder, depression and schizophrenia, is discussed.
Oxidative stress and metabolism in animal model of colitis induced by dextran sulfate sodium
TLDR
The aim of this work was to evaluate the effects of N‐acetylcysteine (NAC) and deferoxamine (DFX) in the treatment of colitis induced by dextran sulfate sodium (DSS).
Methylmalonate administration decreases Na+,K+‐ATPase activity in cerebral cortex of rats
TLDR
Na+, K+-ATPase and Mg2+- ATPase activities were determined in the synaptic plasma membranes from cerebral cortex of rats, and MMA at final concentrations ranging from 0.1 to 2.0 mM had no in vitro effect on these enzyme activities.
Antioxidant treatment reverses organ failure in rat model of sepsis: role of antioxidant enzymes imbalance, neutrophil infiltration, and oxidative stress.
TLDR
Positive correlation between thiobarbituric acid reactive species (TBARS) and markers of organ injury in lung and kidney was found and antioxidant effects seemed to depend not only on the diminution of oxidative damage but also on its anti-inflammatory activity.
Reduction of Na+,K+-ATPase Activity in Hippocampus of Rats Subjected to Chemically Induced Hyperhomocysteinemia
TLDR
The results suggest that the brain dysfunction found in homocystinuria may be related to the reduction of brain Na+,K+-ATPase activity.
Mitochondrial activity and oxidative stress markers in peripheral blood mononuclear cells of patients with bipolar disorder, schizophrenia, and healthy subjects.
TLDR
The results suggest that mitochondrial complex I dysfunction and oxidative stress play important roles in the pathophysiology of SZ and may be used in potential novel adjunctive therapy for SZ, focusing primarily on cognitive impairment and disorder progression.
In vitro effects of silver nanoparticles on the mitochondrial respiratory chain
TLDR
It is demonstrated that AgN (10, 25, and 50 mg l−1) decreases the activity of mitochondrial respiratory chain complexes I, II, III, and IV from all tissues.
In Vitro Effect of Homocysteine on Some Parameters of Oxidative Stress in Rat Hippocampus
TLDR
The results suggest that oxidative stress is involved in the neurological dysfunction of homocystinuria, however, further studies are necessary to confirm and extend the findings to the human condition and also to determine whether antioxidant therapy may be of benefit to these patients.
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