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The anorectic effect of a chronic peripheral infusion of amylin is abolished in area postrema/nucleus of the solitary tract (AP/NTS) lesioned rats
TLDR
Neurons in the AP/NTS region are necessary for chronically elevated peripheral amylin to reduce food intake in rats, and high doses of amyl in may be able to overrun these receptors and reduce feeding by acting at other brain sites.
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Amylin receptors mediate the anorectic action of salmon calcitonin (sCT)
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Amylin decreases meal size in rats
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Comparison of the effects of bacterial lipopolysaccharide and muramyl dipeptide on food intake
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Control of food intake by fatty acid oxidation.
TLDR
Results indicate fatty acid oxidation is involved in the control of food intake, at least when the dietary fat level is relatively high, and mercaptoacetate (MA), an inhibitor of fatty acids oxidation, was studied.
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Evidence for a vagally mediated satiety signal derived from hepatic fatty acid oxidation.
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Reduction of food intake in rats by intraperitoneal injection of low doses of amylin
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Ghrelin Acts on Leptin‐Responsive Neurones in the Rat Arcuate Nucleus
TLDR
Data show that ghrelin interacts with the leptin hypothalamic network in the arcuate nucleus, and may serve as a neurophysiological correlate of the orexigenic and anorectic effects of gh Relin and leptin.
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Control of food intake by fatty acid oxidation and ketogenesis.
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Site-specific effects of ghrelin on the neuronal activity in the hypothalamic arcuate nucleus
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