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The uptake, metabolism, transport and transfer of nitrogen in an arbuscular mycorrhizal symbiosis.
TLDR
Arginine (Arg) was the predominant free AA in the ERM, and almost all Arg molecules became labeled within 3 wk of supplying (15)NH(4) (+) to the fungal compartment, which is the most likely form of N transferred to host cells following its generation from Arg breakdown.
Gastric mucosal apoptosis induced by ethanol: effect of antiulcer agents
TLDR
The results demonstrate that ethanol‐induced gastric epithelial cells apoptosis triggered by the enhancement in mucosal TNF‐α is efficiently counteracted by ebrotidine.
Induction of buccal mucosal apoptosis with chronic alcohol ingestion
TLDR
It is demonstrated that ethanol‐induced buccal mucosal cells apoptosis is triggered by the enhancement in TNF‐α expression, which is a significant increase over that of the controls.
Adrenergic and cholinergic regulation of gastric mucus phospholipid secretion.
TLDR
The results demonstrate the involvement of neural mediators in the regulation of phospholipid secretion in gastric mucus and enhance the influence of beta-adrenergic agonist isoproterenol to a greater extent than by the cholinergic agonists pilocarpine.
Activation of apoptotic caspase‐3 and nitric oxide synthase‐2 in buccal mucosa with chronic alcohol ingestion
TLDR
Results implicate caspase‐3 in the process of alcohol‐induced epithelial cells apoptosis, and point towards participation of NOS‐2 in the amplification of the cell death signaling cascade.
Impact of ethanol on innate protection of gastric mucosal epithelial surfaces and the risk of injury.
TLDR
Ethanol impairs the synthesis of mucin oligosaccharides-dependent mucin-MBP complex formation, and the retention on gastric mucosal surfaces, which is concluded to be the in vitro impact of ethanol on generation of carbohydrate chains in Golgi.
Induction of acute gastritis and epithelial apoptosis by Helicobacter pylori lipopolysaccharide.
TLDR
It is shown that H. pylori lipopolysaccharide induces histologic lesions typical of acute gastritis and that these changes are reflected in the increased epithelial cell apoptosis, and that the apoptotic index showed positive correlation with the grade of the induced inflammatory changes.
Susceptibility of Helicobacter pylori to antimicrobial agents: effect of ebrotidine and ranitidine.
Convincing evidence now exists that infection with H. pylori is a primary factor in the pathogenesis of gastric disease, and new therapy regimens include a combination of H2 blockers with
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