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Carbon nanotubes induce oxidative DNA damage in RAW 264.7 cells
The induction of DNA and chromosome damage following in vitro exposure to carbon nanotubes (CNT) was assessed on the murine macrophage cell line RAW 264.7 by means of the micronucleus (MN) and theExpand
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Biomarkers of oxidative stress after controlled human exposure to ozone.
This study was aimed at evaluating whether controlled short-term exposure to ozone (O(3)) induces changes in biomarkers of lung inflammation and oxidative stress in exhaled breath condensate (EBC)Expand
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Comparative genotoxicity of cobalt nanoparticles and ions on human peripheral leukocytes in vitro.
Owing to the increasing development of nanotechnology, there is a need to assess how engineered nanomaterials can interact with living cells. The main purpose of the present study was to assessExpand
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Serum clara cell protein: a sensitive biomarker of increased lung epithelium permeability caused by ambient ozone.
Ozone in ambient air may cause various effects on human health, including decreased lung function, asthma exacerbation, and even premature mortality. These effects have been evidenced using variousExpand
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Nephropathies and exposure to perchloroethylene in dry-cleaners
Even in specific risk groups, the relation between exposure to organic solvents and chronic renal diseases remains controversial. Thus, in a collaborative European study, we assessed the renalExpand
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Polymorphism of quinone-metabolizing enzymes and susceptibility to ozone-induced acute effects.
The role of the genetic polymorphism of NAD(P)H:quinone oxidoreductase (NQO1) and glutathione-S-transferase micro-1 (GSTM1) in the responsiveness to O(3)-induced acute effects was investigated in 24Expand
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Polymorphism of xenobiotic-metabolizing enzymes and excretion of styrene-specific mercapturic acids.
The role of polymorphic xenobiotic-metabolizing enzymes in the interindividual variability of phenylhydroxyethyl mercapturic acids (PHEMAs) was investigated in 56 styrene-exposed workers. AmbientExpand
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Experimental model of lead nephropathy. I. Continuous high-dose lead administration.
This study followed the progression of lead nephropathy in male Sprague-Dawley rats (E) administered lead acetate (0.5%) continuously in drinking water for periods ranging from 1 to 12 months.Expand
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Experimental model of lead nephropathy. II. Effect of removal from lead exposure and chelation treatment with dimercaptosuccinic acid (DMSA).
Male Sprague-Dawley rats were exposed to high-dose (0.5%) lead acetate for periods ranging from 1 to 9 months; then lead exposure was discontinued, and animals were sacrificed after 12 months.Expand
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Motor function, olfactory threshold, and hematological indices in manganese-exposed ferroalloy workers.
A cross-sectional study was conducted in 35 male subjects randomly selected from workers of a ferroalloy production plant and exposed to manganese (Mn) oxides; the objective was to detect early signsExpand
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