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Cardiac Troponin T and Hydration Status as Prognostic Markers in Hemodialysis Patients
TLDR
Longer dialysis vintage associates with CV dysfunction, overhydration and increased mortality, which may be predicted with OH% and cTnT.
N‑terminal pro‑B-type natriuretic peptide as a marker of hypervolemia and predictor of increased mortality in patients on hemodialysis.
TLDR
NT‑proBNP seems to be a useful biomarker of hypervolemia in HD patients, Nevertheless, it has to be interpreted with regard to the patient's individual residual renal function and cardiovascular status.
ASSUMPTIONS OF MAXIMAL INDIVIDUALISM IN THE TIME OF THE COVID-19 PANDEMIC: ETHICAL ASPECTS
Maximal individualism, which is currently a prevalent trend in the way many patients think, places high hopes in the achievements of biomedicine and assumes that everyone should always receive
Hemodialysis‐induced changes in the blood composition affect function of the endothelium
TLDR
Hemodialysis‐induced intravascular inflammation changes the function of endothelial cells, which may lead to acceleration of atherosclerosis.
Sulodexide suppresses inflammation in patients with chronic venous insufficiency.
TLDR
Treatment with SUL in patients with CVI reduces intravascular inflammation and is protective for the endothelial cells and for the extracellular matrix changes related to metalloproteinase expression.
N‐Acetylglucosamine modulates function of the skin fibroblasts
Fibroblasts are an important component of the skin determining its properties. N‐Acetylglucosamine (NAG) is the substrate for hyaluronan synthesis, and it also has anti‐inflammatory and
Sulodexide Reduces the Proinflammatory Effect of Serum from Patients with Peripheral Artery Disease in Human Arterial Endothelial Cells
TLDR
Atherosclerotic serum induces proinflammatory and prothrombotic phenotype in arterial endothelium, which is partially reduced by sulodexide, via inhibition of genes expression, and in consequence lower secretory activity.
Sulodexide modifies intravascular homeostasis what affects function of the endothelium.
TLDR
Sulodexide reduces intravascular inflammation and suppresses inflammatory reaction in the endothelial cells; both effects are desirable in patients with peripheral vascular disease.
Sulodexide reduces the inflammatory reaction and senescence of endothelial cells in conditions involving chronic venous disease.
TLDR
Chronic in vitro exposure of human venous endothelial cells to medium supplemented with CVD patient serum induces an inflammatory phenotype and slows HUVEC senescence in the milieu of CVD serum.
Normal saline may promote formation of peritoneal adhesions.
TLDR
Mesothelial cells exposed in vivo to normal saline dwelling in the abdominal cavity acquire properties which may accelerate formation of the peritoneal adhesions.
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