• Publications
  • Influence
Association between genetic polymorphisms in the XRCC1, XRCC3, XPD, GSTM1, GSTT1, MSH2, MLH1, MSH3, and MGMT genes and radiosensitivity in breast cancer patients.
TLDR
This is the first report to find an association between MSH2 and MSH3 genetic variants and the development of radiosensitivity in breast cancer patients, and suggests the hypothesis that mismatch repair mechanisms may be involved in cellular response to radiotherapy. Expand
Cell Invasion Is Affected by Differential Expression of the Urokinase Plasminogen Activator/Urokinase Plasminogen Activator Receptor System in Muscle Satellite Cells from Normal and Dystrophic
TLDR
It is shown that normal MSC are more prone than DMD cells to spontaneous invasion but, when subjected to a chemotactic gradient of u-PA, DMD MSC sense the ligand much better and to a greater extent thannormal MSC. Expand
The Role of Apoptosis in the Pathogenesis of Dermatitis Herpetiformis
TLDR
It is concluded that in DH, as well as in BP, apoptosis plays a role in the pathogenesis of cutaneous lesions in concert with other pathogenetic mechanisms. Expand
Integrin-mediated neurite outgrowth in neuroblastoma cells depends on the activation of potassium channels
TLDR
Electrical signals elicited by integrin interaction with ECM components and their role in neurite outgrowth were studied in two clones isolated from 41A3 murine neuroblastoma cell line, revealing a similar implication of integrins in the adhesion of these clones to the ECM proteins. Expand
Expression of cytokines and chemokine receptors in the cutaneous lesions of erythema multiforme and Stevens–Johnson syndrome/toxic epidermal necrolysis
TLDR
This work has shown that Erythema multiforme and Stevens–Johnson syndrome/toxic epidermal necrolysis are caused by a dysregulation of cellular immunity. Expand
The CD40/CD40 ligand system is expressed in the cutaneous lesions of erythema multiforme and Stevens–Johnson syndrome/toxic epidermal necrolysis spectrum
TLDR
This work has shown that Erythema multiforme and Stevens–Johnson syndrome/toxic epidermal necrolysis are determined by a dysregulation of cellular immunity. Expand
Interaction of tumor cells with vascular endothelia: Role of platelet-activating factor
TLDR
It is demonstrated that PAF-mediated activation of tumor cells enhances their adhesiveness to IL-1-stimulated vascular endothelia and protein tyrosine phosphorylation was observed in Hs294T and LS180 cells stimulated by PAF. Expand
FGF2‐mediated upregulation of urokinase‐type plasminogen activator expression requires a MAP‐kinase dependent activation of poly(ADP‐ribose) polymerase
TLDR
It is demonstrated that specific inhibition of poly(ADP‐ribose) polymerase activity via 3‐aminobenzamide (3ABA) or NAD+ deprivation prevents FGF2‐mediated uPA mRNA over‐expression and cell‐associated plasminogen activator (PA) production in GM7373 endothelial cell line. Expand
FoxP3-expressing T regulatory cells in atopic dermatitis lesions.
TLDR
Data show that an impaired function of natural T regs may not play a primary role in the pathophysiology of AD lesions, and healthy skin specimens featured few CD25+ FoxP3+ cells scattered throughout the dermis. Expand
Asthma and respiratory symptoms in 6-7 yr old Italian children: gender, latitude, urbanization and socioeconomic factors. SIDRIA (Italian Studies on Respiratory Disorders in Childhood and the
  • E. Barletta
  • Medicine
  • The European respiratory journal
  • 1997
TLDR
Urbanization and socioeconomic level have little effect on the prevalence of wheezing in this area, but they might influence the diagnosis and the management of asthma, as well as the prevalenceof chronic cough and chronic phlegm. Expand
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