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BACKGROUND Hypokalemia increases the vulnerability to ventricular fibrillation. We hypothesize that the apamin-sensitive small-conductance calcium-activated potassium current (IKAS) is activated during hypokalemia and that IKAS blockade is proarrhythmic. METHODS AND RESULTS Optical mapping was performed in 23 Langendorff-perfused rabbit ventricles with(More)
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