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Immunolocalization of estrogen receptor beta in the mouse brain: comparison with estrogen receptor alpha.
Estrogen receptor alpha (ER alpha) and ER beta are members of the steroid nuclear receptor family that modulate gene transcription in an estrogen-dependent manner. ER mRNA and protein have beenExpand
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Immunolocalization of estrogen receptor β in the mouse brain: Comparison with estrogen receptor α
Estrogen receptor α (ERα) and ERβ are members of the steroid nuclear receptor family that modulate gene transcription in an estrogen-dependent manner. ER mRNA and protein have been detected bothExpand
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Origin of luteinizing hormone-releasing hormone neurons
NEURONS expressing luteinizing hormone-releasing hormone (LHRH), found in the septal-preoptic nuclei and hypothalamus1–3, control the release of gonadotropic hormones from the anterior pituitaryExpand
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Assessing the molecular genetics of attention networks
BackgroundCurrent efforts to study the genetic underpinnings of higher brain functions have been lacking appropriate phenotypes to describe cognition. One of the problems is that many cognitiveExpand
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Stomach ghrelin-secreting cells as food-entrainable circadian clocks
Increases in arousal and activity in anticipation of a meal, termed “food anticipatory activity” (FAA), depend on circadian food-entrainable oscillators (FEOs), whose locations and output signalsExpand
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Long-term gene expression and phenotypic correction using adeno-associated virus vectors in the mammalian brain
Adeno-associated viral (AAV) vectors are non-pathogenic, integrating DNA vectors in which all viral genes are removed and helper virus is completely eliminated. To evaluate this system in theExpand
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Estrogens and brain function
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An estrogen-dependent four-gene micronet regulating social recognition: A study with oxytocin and estrogen receptor-α and -β knockout mice
Estrogens control many physiological and behavioral processes, some of which are connected to reproduction. These include sexual and other social behaviors. Here we implicate four gene products in aExpand
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Luteinizing hormone-releasing hormone (LHRH)-expressing cells do not migrate normally in an inherited hypogonadal (Kallmann) syndrome.
Kallmann syndrome inherited hypogonadotropic hypogonadism with anosmia, is associated with an X-chromosome deletion at Xp 22.3. In a Kallmann fetus, we have found an absence of luteinizingExpand
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Silencing of estrogen receptor α in the ventromedial nucleus of hypothalamus leads to metabolic syndrome
Estrogen receptor α (ERα) plays a pivotal role in the regulation of food intake and energy expenditure by estrogens. Although it is well documented that a disruption of ERα signaling in ERα knockoutExpand
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