• Publications
  • Influence
Sleep deprivation induces brain region‐specific decreases in glutathione levels
Results indicate that specific brain areas may be differentially susceptible to oxidative stress after sleep deprivation and the apparent vulnerability of the hypothalamus to these effects may contribute to the functional effects of sleep deprivation.
Differential propensity to ethanol sensitization is not associated with altered binding to D1 receptors or dopamine transporters in mouse brain
It is suggested that brain D1 and DAT binding, unlike the recently reported changes in D2 binding, do not differentiate mice that develop behavioral sensitization to ethanol from those that do not.
Increased ACTH and corticosterone secretion induced by different methods of paradoxical sleep deprivation
The results indicate that the multiple platform technique induces a distinct activation of the HPA axis, and that PS deprivation may act as an additional stressor.
Sensitization to ethanol’s stimulant effect is associated with region-specific increases in brain D2 receptor binding
The observed increases in D2-receptor binding in circumscribed targets of nigrostriatal projections may reflect either a pre-existing condition in sensitization-prone animals or a selective vulnerability of D2 receptors to chronic EtOH in these animals, and may be a marker for differential susceptibility to EtOH sensitization.
Paradoxical Sleep Deprivation and Sleep Recovery: Effects on the Hypothalamic–Pituitary–Adrenal Axis Activity, Energy Balance and Body Composition of Rats
Results showed that sleep deprivation produces increased energy expenditure and loss of fat content, and most of the alterations were reversed by sleep recovery, except for corticosterone levels and body composition.