Christopher Badcock

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Autistic-spectrum conditions and psychotic-spectrum conditions (mainly schizophrenia, bipolar disorder, and major depression) represent two major suites of disorders of human cognition, affect, and behavior that involve altered development and function of the social brain. We describe evidence that a large set of phenotypic traits exhibit diametrically(More)
We describe a new hypothesis for the development of autism, that it is driven by imbalances in brain development involving enhanced effects of paternally expressed imprinted genes, deficits of effects from maternally expressed genes, or both. This hypothesis is supported by: (1) the strong genomic-imprinting component to the genetic and developmental(More)
The imprinted brain theory proposes that autism spectrum disorder (ASD) represents a paternal bias in the expression of imprinted genes. This is reflected in a preference for mechanistic cognition and in the corresponding mentalistic deficits symptomatic of ASD. Psychotic spectrum disorder (PSD) would correspondingly result from an imbalance in favor of(More)
nesses such as schizophrenia and autism tend to run in families. But neither disorder obeys classical Mendelian laws of inheritance, making it difficult to pinpoint the genes involved. We believe that psychiatric illness may be less to do with the genes a mother and father pass down, and more to do with which genes they program for expression. By our(More)
Inclusive fitness theory predicts phenotypic conflicts between the effects of genes that differ in their means of increased replication. Such conflicts may involve distinct modes of inheritance or different likelihoods of genes identical by descent being present in interacting individuals, such as for autosomal genes in various classes of relative.(More)
Sociological Notes No. 5 ISSN 0267 7113 ISBN 1 870614 70 4 An occasional publication of the Libertarian Alliance, 25 Chapter Chambers, Esterbrooke Street, London SW1P 4NN email: © 1990: Libertarian Alliance; London School of Economics Quarterly; Christopher Badcock. This article first appeared in The London(More)
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