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  • John W Calvert, Marah E. Condit, +8 authors David J. Lefer
  • Chemistry, Medicine
  • Circulation research
  • 2011 (First Publication: 10 June 2011)
  • Rationale: Exercise training confers sustainable protection against ischemia–reperfusion injury in animal models and has been associated with improved survival following a heart attack in humans. ItExpand
  • Adrienne L. King, David J. Polhemus, +13 authors David J. Lefer
  • Medicine, Chemistry
  • Proceedings of the National Academy of Sciences
  • 2014 (First Publication: 10 February 2014)
  • Significance Physiological concentrations of hydrogen sulfide (H2S) exert potent prosurvival actions. We demonstrate that the cytoprotective actions of H2S are mediated in part via a second gaseousExpand
  • Bridgette F. Peake, Chad K. Nicholson, +4 authors John W Calvert
  • Medicine
  • American journal of physiology. Heart and…
  • 2013 (First Publication: 1 May 2013)
  • Hydrogen sulfide (H2S) therapy protects nondiabetic animals in various models of myocardial injury, including acute myocardial infarction and heart failure. Here, we sought to examine whether H2SExpand
  • Larry A. Barr, Yuuki Shimizu, Jonathan P. Lambert, Chad K. Nicholson, John W Calvert
  • Medicine
  • Nitric oxide : biology and chemistry
  • 2015 (First Publication: 30 April 2015)
  • Diabetic cardiomyopathy is a significant contributor to the morbidity and mortality associated with diabetes and metabolic syndrome. However, the underlying molecular mechanisms that lead to itsExpand
  • Jonathan P. Lambert, Chad K. Nicholson, Hena Amin, Sana Abida Amin, John W Calvert
  • Medicine
  • Medical gas research
  • 2014 (First Publication: 12 December 2014)
  • BackgroundCoronary artery disease remains the principal cause of death in patients with diabetes mellitus. Diabetic mice display exacerbated injury following myocardial ischemia-reperfusion (MI/R)Expand
  • Shashi Bhushan, Kazuhisa Kondo, +9 authors David J. Lefer
  • Medicine
  • Circulation research
  • 2014 (First Publication: 11 April 2014)
  • Rationale: Nitric oxide (NO) bioavailability is reduced in the setting of heart failure. Nitrite (NO2) is a critically important NO intermediate that is metabolized to NO during pathological states.Expand