Learn More
Rabbits were continuously exposed to 200 ppm carbon monoxide. Using the same criteria as applied by earlier investigators for morphological myocardial damage, no histotoxic effect on myocardial morphology could be demonstrated when electron-microscopic investigations were performed blindly. Similarly, exposure to 0.5 ppm hydrogen cyanide, 0.5 ppm hydrogen(More)
In 24 kindergartens the relation between the presence of readily visible, man-made mineral-fiber products in the ceilings and the presence/frequency of symptoms or diseases and the correlations between complaints of symptoms or disease and the concentration of man-made mineral-fibres in the indoor environment was investigated. A combination of traditional(More)
  • C Hugod
  • Archives of environmental health
  • 1979
Four male rabbits were continuously exposed to 5 ppm nitric oxide for 14 days. Four rabbits served as controls. Specimens from pulmonary arterioles and alveolar tissue were examined by light and transmission electron microscopy. The most conspicuous findings were fluid-containing vacuoles inside the arteriolar endothelial cells and/or in the intercellular(More)
Rabbits were continously exposed either to 200 parts per million carbon monoxide for 3, 4, and 6 weeks or to 1,900 parts per million carbon monoxide for 5 h. Blind light and transmission electron microscopic investigations showed no morphological changes in the lung parenchyma, lung vasculature or in pulmonary arteries of the exposed animals when compared(More)
Non-cholesterol-fed rabbits were exposed to carbon monoxide at concentrations in air of either 200, 2000, or 4000 parts per million (=0.02, 0.2 or 0.4%, vol/vol). Using the same criteria for intimal damage as in earlier morphological studies, no histotoxic effect on intimal/subintimal morphology of coronary arteries or the aorta could be demonstrated, when(More)