• Publications
  • Influence
Homophobia? No, homoprejudice.
  • C. R. Logan
  • Psychology, Biology
    Journal of homosexuality
  • 1996
TLDR
Findings indicated that there is little, if any, evidence to support the characterization of anti-homosexual responses as a phobia and that males were less tolerant than females of gays and lesbians and males and females respond differentially according to same-sex target.
Studies on lysergic acid diethylamide (LSD-25). I. Effects in former morphine addicts and development of tolerance during chronic intoxication.
TLDR
The striking mental changes induced by the diethylamide of lysergic acid (hereafter referred to as LSD) have been studied extensively in Europe, Great Britain and the United States and are apparently the most effective and safest agent for inducing an experimental, but reversible, psychosis in nonpsychotic subjects.
The effect of N,N-dimethyltryptamine in human subjects tolerant to lysergic acid diethylamide
TLDR
The spectrum of effects produced with single doses of N,N-dimethyltryptamine in 6 nontolerant human subjects resembled those produced with lysergic acid diethylamide (LSD), suggesting that the site or mechanism which is altered during LSD tolerance is not one which is primarily concerned with the action of DMT.
The respiratory effects of morphine during a cycle of dependence.
TLDR
The data indicate that dependence on morphine produces both an early (primary) and protracted (secondary) abstinence syndrome in man, consistent with the homeostatic and redundancy theory of physical dependence.
Studies on the diethylamide of lysergic acid (LSD-25). II. Effects of chlorpromazine, azacyclonol, and reserpine on the intensity of the LSD-reaction.
TLDR
The purpose of the present paper is to present the results of experiments in which attempts were made to block (prevent) or reverse (treat) the LSD reaction with chlorpromazine, azacyclonol (Frenquel), and reserpine.
Studies on lysergic acid diethylamide (LSD-25). III. Attempts to attenuate the LSD-reaction in man by pretreatment with neurohumoral blocking agents.
TLDR
A hypothesis which ascribes the LSD psychosis to competition between LSD and serotonin for receptor sites on or in neurons, which might be termed the serotonin-deficiency theory, is based in part on the following evidence: Serotonin is found in brain, 6.
Relationships of psychotomimetic to anti-serotonin potencies of congeners of lysergic acid diethylamide (LSD-25)
TLDR
The data do not support but do not disprove the “serotonin deficiency” hypothesis of the LSD psychosis, and high potency as a serotonin antagonist in isolated smooth muscle preparations was not correlated with high potency in man as a psychotomimetic.
Cross tolerance between d-2-brom-lysergic acid diethylamide (BOL-148) and the d-diethylamide of lysergic acid (LSD-25)
TLDR
Simultaneous administration of 2 to 4 mg of d-2-Brom lysergic acid diethylamide (BOL-148) did not reduce the intensity of the reaction caused by 0.5 to 1.5mcg/kg of LSD-25, and pre-treatment resulted in statistically significant attenuation of all aspects of the LSD reaction that were measured.