Share This Author
GH, GH receptor, GH secretagogue receptor, and ghrelin expression in human T cells, B cells, and neutrophils.
- N. Hattori, T. Saito, T. Yagyu, B. Jiang, K. Kitagawa, C. Inagaki
- Biology, MedicineJournal of Clinical Endocrinology and Metabolism
- 1 September 2001
Widespread distribution of ghrelin and GH secretagogue receptor in human immune cells may indicate unknown biological functions other than enhancing GH secretion in the immune system.
Long‐Term Treatment with Antidepressants Increases Glucocorticoid Receptor Binding and Gene Expression in Cultured Rat Hippocampal Neurones
- Okugawa, Omori, Suzukawa, Fujiseki, T. Kinoshita, C. Inagaki
- Biology, PsychologyJournal of neuroendocrinology
- 1 November 1999
It is suggested that tricyclic antidepressants directly increase hippocampal GR by short‐term and long-term exposure, and that the increase by long‐term exposure is evoked commonly with different classes of antidepressants through transcriptional up‐regulation of GR expression.
Novel microsomal anion-sensitive Mg2+-ATPase activity in rat brain.
A Cl- pump in rat brain neurons.
In cultured hippocampal pyramidal cell-like neurons from embryonic rat brain, ethacrynic acid and ATP-consuming treatment increased, but furosemide, an inhibitor of Na+/K+/Cl- cotransporter, decreased, [Cl-]i when monitored using Cl(-)-sensitive fluorescent probes, suggesting that region-specific localization and developmental changes in the activities of Cl- transporters probably result in uneven and age-dependent distribution in the neurons.
Anti-prolactin (PRL) autoantibodies cause asymptomatic hyperprolactinemia: bioassay and clearance studies of PRL-immunoglobulin G complex.
The data suggest that PRL forms a complex with IgG, and this probably results in delayed clearance of PRL and leads to hyperprolactinemia in women with anti-PRL autoantibodies.
The activities of noradrenergic and dopaminergic neuron systems in experimental hydrocephalus.
In experimental hydrocephalus in the rabbit, NA release is increased throughout the brain, while DA release is decreased in the cerebral cortex and caudate nucleus, and increased in the cerebellum, hypothalamus, midbrain, and pons plus medulla oblongata.
CI-ATPase and Na+/K(+)-ATPase activities in Alzheimer's disease brains.
It is demonstrated that Cl(-)-ATPase and Na+/K(+)- ATPase are selectively impaired in AD brains, which may reduce the gradients of Na(+), K(+) and Cl(-) across the cell membranes to cause excitotoxic cellular response and the resulting neuronal death.
An ATP-driven Cl− pump regulates Cl− concentrations in rat hippocampal neurons
Uneven distribution of intracellular Cl− in rat hippocampal neurons