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A clinical approach to diagnosis of autoimmune encephalitis
Potassium channel antibody-associated encephalopathy: a potentially immunotherapy-responsive form of limbic encephalitis.
VGKC-Ab-associated encephalopathy is a relatively common form of autoimmune, non-paraneoplastic, potentially treatable encephalitis that can be diagnosed by a serological test.
Pathogenesis, diagnosis and treatment of Rasmussen encephalitis: a European consensus statement.
This work proposes formal diagnostic criteria and a therapeutic pathway for the management of RE patients, and summarizes recent data on the pathogenesis, clinical and paraclinical presentation, and therapeutic approaches.
N-methyl-d-aspartate antibody encephalitis: temporal progression of clinical and paraclinical observations in a predominantly non-paraneoplastic disorder of both sexes
Overall, the data support a model in which the early features of N-methyl-d-aspartate receptor encephalopathy are associated with cerebrospinal fluid lymphocytosis, and the later features with appearance of oligoclonal bands, which is associated with restriction to the first stage.
Autoantibodies associated with diseases of the CNS: new developments and future challenges
Antibodies to glutamic acid decarboxylase define a form of limbic encephalitis
- M. Malter, C. Helmstaedter, H. Urbach, A. Vincent, C. Bien
- Medicine, PsychologyAnnals of neurology
- 1 April 2010
A cohort of patients with recent‐onset temporal lobe epilepsy caused by LE was studied to test for GAD antibody positivity and response to immunotherapies.
International consensus classification of hippocampal sclerosis in temporal lobe epilepsy: A Task Force report from the ILAE Commission on Diagnostic Methods
The proposed international consensus classification of hippocampal neuronal cell loss will aid in the characterization of specific clinicopathologic syndromes, and explore variability in imaging and electrophysiology findings, and in postsurgical seizure control.
The natural history of Rasmussen's encephalitis.
It is demonstrated that most of the brain damage in RE occurs during the first 8-12 months, and these findings should be taken into consideration when future therapeutic approaches to RE are evaluated.
Immunopathology of autoantibody-associated encephalitides: clues for pathogenesis.
Findings strongly support a central role for T cell-mediated neuronal cytotoxicity in encephalitides with antibodies against intracellular antigens in voltage-gated potassium channel-complex encephalitis.
Diagnosis and staging of Rasmussen’s encephalitis by serial MRI and histopathology
A disease course with the highest inflammatory intensity in the early stages and a subsequent decrease in inflammation is suggested in patients with Rasmussen’s encephalitis.