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  • Michael Hirth, Roman Rukwied, Alois Gromann, Brian P. Turnquist, Otilia Obreja
  • Medicine
  • PAIN®
  • 2013 (First Publication: 1 November 2013)
  • Summary Three weeks after nerve growth factor injection in pigs we found nociceptor sensitization possibly underlying NGF‐evoked hyperalgesia in human skin without evidence for nociceptor sprouting.Expand
  • Otilia Obreja, Matthias Ringkamp, Brian P. Turnquist, Michael Hirth, Martin Schmelz
  • Medicine
  • PAIN
  • 2011 (First Publication: 1 September 2011)
  • Summary Intracutaneous nerve growth factor reduces the activity‐dependent slowing of conduction of the mechano‐insensitive C‐fibers. This long‐term change of axonal characteristics might contributeExpand
  • Wolfgang G. Schneider, Robert M Slugg, Brian P. Turnquist, Richard A. Meyer, John N. Campbell
  • Biology, Medicine
  • Journal of Neuroscience Methods
  • 1995 (First Publication: 1 August 1995)
  • We have developed a computer-based electromechanical stimulator system suited for neurophysiological and psychophysical studies of pain. The core of the stimulator is a servo-controlled linear motorExpand
  • Christian König, Eric Morch, +4 authors Andrea Ebersberger
  • Medicine
  • The Journal of Neuroscience
  • 2016 (First Publication: 21 September 2016)
  • During peripheral inflammation, both spinal TNF-α and IL-6 are released within the spinal cord and support the generation of inflammation-evoked spinal hyperexcitability. However, whether spinalExpand
  • Amanda H Klein, Alina Vyshnevska, +8 authors Matthias Ringkamp
  • Medicine, Biology
  • The Journal of Neuroscience
  • 2017 (First Publication: 17 May 2017)
  • Voltage-gated sodium (NaV) channels are responsible for the initiation and conduction of action potentials within primary afferents. The nine NaV channel isoforms recognized in mammals are oftenExpand
  • Otilia Obreja, Michael Hirth, Brian P. Turnquist, Roman Rukwied, Matthias Ringkamp, Martin Schmelz
  • Medicine
  • Anesthesia and analgesia
  • 2012 (First Publication: 1 May 2012)
  • BACKGROUND: Axonal sodium channels are attractive targets for chronic pain treatment, and recent evidence suggests that specific targeting of the slow inactivation of sodium channels (NaV) mightExpand