Blanca Heras-Pozas

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Activation of pancreatic β-cell proliferation has been proposed as an approach to replace reduced functional β-cell mass in diabetes. Quiescent fibroblasts exit from G0 (quiescence) to G1 through pRb phosphorylation mediated by cyclin C/cdk3 complexes. Overexpression of cyclin D1, D2, D3, or cyclin E induces pancreatic β-cell proliferation. We hypothesized(More)
Margarita Jiménez-Palomares,* José Francisco López-Acosta,* Pablo Villa-Pérez, José Luis Moreno-Amador, Jennifer Muñoz-Barrera, Sara Fernández-Luis, Blanca Heras-Pozas, Germán Perdomo, Ernesto Bernal-Mizrachi, and Irene Cózar-Castellano Instituto de Genética y Biología Molecular, Universidad de Valladolid-CSIC, Valladolid, Spain; Division of Metabolism,(More)
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