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Diagnostic criteria for multiple sclerosis: 2010 Revisions to the McDonald criteria
These revisions simplify the McDonald Criteria, preserve their diagnostic sensitivity and specificity, address their applicability across populations, and may allow earlier diagnosis and more uniform and widespread use.
Diagnostic criteria for multiple sclerosis: 2005 revisions to the “McDonald Criteria”
New evidence and consensus now strengthen the role of these criteria in the multiple sclerosis diagnostic workup to demonstrate dissemination of lesions in time, to clarify the use of spinal cord lesions, and to simplify diagnosis of primary progressive disease.
A serum autoantibody marker of neuromyelitis optica: distinction from multiple sclerosis
The clinical course of neuromyelitis optica (Devic’s syndrome)
Clinical, laboratory, and imaging features generally distinguish neuromyelitis optica from MS, and patients with relapsing optic neuritis and myelitis may have neuromyeliitis opticas rather than MS.
The spectrum of neuromyelitis optica
International consensus diagnostic criteria for neuromyelitis optica spectrum disorders
The International Panel for NMO Diagnosis (IPND) was convened to develop revised diagnostic criteria using systematic literature reviews and electronic surveys to facilitate consensus and achieved consensus on pediatric NMOSD diagnosis and the concepts of monophasicNMOSD and opticospinal MS.
Diagnosis of multiple sclerosis: 2017 revisions of the McDonald criteria
Revised diagnostic criteria for neuromyelitis optica
- D. Wingerchuk, V. Lennon, S. Pittock, C. Lucchinetti, B. Weinshenker
- Medicine, PsychologyNeurology
- 23 May 2006
Revised diagnostic criteria for definite neuromyelitis optica (NMO) that require optic neuritis, myelitis, and at least two of three supportive criteria: MRI evidence of a contiguous spinal cord lesion 3 or more segments in length, onset brain MRI nondiagnostic for multiple sclerosis, or NMO-IgG seropositivity.
A role for humoral mechanisms in the pathogenesis of Devic's neuromyelitis optica.
The extent of complement activation, eosinophilic infiltration and vascular fibrosis observed in the Devic NMO cases is more prominent compared with that in prototypic multiple sclerosis, and supports a role for humoral immunity in the pathogenesis of NMO.