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UNIDENTIFIED CURVED BACILLI IN THE STOMACH OF PATIENTS WITH GASTRITIS AND PEPTIC ULCERATION
TLDR
The bacteria were present in almost all patients with active chronic gastritis, duodenal ulcer, or gastric ulcer and thus may be an important factor in the aetiology of these diseases.
UNIDENTIFIED CURVED BACILLI ON GASTRIC EPITHELIUM IN ACTIVE CHRONIC GASTRITIS
TLDR
Gastric biopsy findings are classified according to the type of inflammation, regardless of other features, as "no inflammation", "chronic gastritis", or "active chronic gastritis" (ACG).
Campylobacter pyloridis, gastritis, and peptic ulceration.
TLDR
Histological examination of gastric biopsy tissue has shown that C pyloridis is strongly associated with active chronic gastritis, and is not found on normal mucosa except when a biopsy specimen from elsewhere in the stomach shows active chronic Gastritis.
Attempt to fulfil Koch's postulates for pyloric Campylobacter
TLDR
It is proposed that this disorder may progress to a chronic infection which predisposes to peptic ulceration and be linked to acute pyloric Campylobacter gastritis.
The Peopling of the Pacific from a Bacterial Perspective
TLDR
The hpSahul populations in New Guinea and Australia have diverged sufficiently to indicate that they have remained isolated for the past 23,000 to 32,000 years, and the second human expansion from Taiwan 5000 years ago dispersed one of several subgroups of the Austronesian language family into Melanesia and Polynesia.
Pyloric Campylobacter infection and gastroduodenal disease
TLDR
It is suggested that pyloric Campylobacter infection is a major factor in the causation of dyspeptic disease and peptic ulceration and antibacterial regimens directed against the bacterium may provide a permanent cure for these chronic disorders.
Urea protects Helicobacter (Campylobacter) pylori from the bactericidal effect of acid.
TLDR
The high urease activity of H. pylori enables it to survive in gastric acid, and the addition of 5 mmol/L of urea completely protected H.pylori from acid.
PROSPECTIVE DOUBLE-BLIND TRIAL OF DUODENAL ULCER RELAPSE AFTER ERADICATION OF CAMPYLOBACTER PYLORI
100 consecutive patients with both duodenal ulcer and Campylobacter pylori infection were followed up to see whether eradication of C pylori affected ulcer healing or relapse. Patients were randomly
Helicobacter pylori: 20 years on.
TLDR
During the typical lifelong chronic infection, two important diseases occur: H. pylori alters gastric physiology to cause acid hypersecretion and peptic ulcer and damages the acid secreting mucosa leading to atrophic gastritis and gastric cancer risk.
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