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Hypertrophic cardiomyopathy: a systematic review.
TLDR
An appreciation that HCM, although an important cause of death and disability at all ages, does not invariably convey ominous prognosis and is compatible with normal longevity should dictate a large measure of reassurance for many patients. Expand
Contemporary definitions and classification of the cardiomyopathies: an American Heart Association Scientific Statement from the Council on Clinical Cardiology, Heart Failure and Transplantation
TLDR
The present classification scheme recognizes the rapid evolution of molecular genetics in cardiology, as well as the introduction of several recently described diseases, and is unique in that it incorporates ion channelopathies as a primary cardiomyopathy. Expand
Sudden death in young competitive athletes. Clinical, demographic, and pathological profiles.
TLDR
Sudden death in young competitive athletes usually is precipitated by physical activity and may be due to a heterogeneous spectrum of cardiovascular disease, most commonly hypertrophic cardiomyopathy. Expand
Task Force on Sudden Cardiac Death of the European Society of Cardiology.
TLDR
The members of the Task Force on Sudden Death dedicate this paper to the memory of their former friend and colleague, Professor Ronald W. F. Campbell, who spent his life working in the field of sudden cardiac death, and the extent of the expanding knowledge in this field is described. Expand
Clinical profile of congenital coronary artery anomalies with origin from the wrong aortic sinus leading to sudden death in young competitive athletes.
TLDR
Standard testing with ECG under resting or exercise conditions is unlikely to provide clinical evidence of myocardial ischemia and would not be reliable as screening tests in large athletic populations, and premonitory cardiac symptoms not uncommonly occurred shortly before sudden death, suggesting that a history of exertional syncope or chest pain requires exclusion of this anomaly. Expand
Congenital heart disease caused by mutations in the transcription factor NKX2-5.
TLDR
Data indicate that NKX2-5 is important for regulation of septation during cardiac morphogenesis and for maturation and maintenance of atrioventricular node function throughout life. Expand
2011 ACCF/AHA guideline for the diagnosis and treatment of hypertrophic cardiomyopathy: a report of the American College of Cardiology Foundation/American Heart Association Task Force on Practice
TLDR
This paper aims to provide a history of FACC/FAHA accreditation practices in the United States from 1989 to 2002, a period chosen in order to explore its role in the development of post-secondary education. Expand
Prevalence of hypertrophic cardiomyopathy in a general population of young adults. Echocardiographic analysis of 4111 subjects in the CARDIA Study. Coronary Artery Risk Development in (Young) Adults.
TLDR
These unique population-based data will aid in assessments of the impact of HCM-related mortality and morbidity in the general population and the practicality of screening large populations for HCM, including those comprising competitive athletes. Expand
Recommendations and Considerations Related to Preparticipation Screening for Cardiovascular Abnormalities in Competitive Athletes: 2007 Update: A Scientific Statement From the American Heart
TLDR
The increasing awareness that automated external defibrillators may not always prove successful in the secondary prevention of sudden death for athletes with cardiovascular disease underscores the importance of preparticipation screening for the prospective identification of at-risk athletes and the prophylactic prevention of cardiac events during sports by selective disqualification. Expand
Natural history and expansive clinical profile of stress (tako-tsubo) cardiomyopathy.
TLDR
The clinical spectrum was heterogeneous with about one-third either male, <or=50 years of age, without a stress trigger, or with in-hospital death, nonfatal recurrence, embolic stroke, or delayed normalization of ejection fraction, and Beta-blocking drugs were not absolutely protective and SC was a marker for increased noncardiac mortality. Expand
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