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Shrinkage-induced Activation of the Na+/H+ Exchanger in Ehrlich Ascites Tumor Cells: Mechanisms Involved in the Activation and a Role for the Exchanger in Cell Volume Regulation
Abstract. Amiloride-sensitive, Na+-dependent, DIDS-insensitive cytoplasmic alkalinization is observed after hypertonic challenge in Ehrlich ascites tumor cells. This was assessed using theExpand
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Linear increase in glycolytic activity through the cell cycle of schizosaccharomyces pombe.
The glycolytic activity of 3 different synchronous system in S. pombe was studied. Synchronous cultures were produced by a selection procedure, by cyclic heat treatment, or by cloning cells from anExpand
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Synchronization of cell divisions in the fission yeast, Schizosaccharomyces pombe, using heat shocks.
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Volume regulation in human fibroblasts: role of Ca2+ and 5-lipoxygenase products in the activation of the Cl− efflux
Trypsinized human skin fibroblasts in suspension perform regulatory volume decrease (RVD) after cell swelling in hypotonic medium. During RVD, 36Cl− efflux is dramatically increased and the cellExpand
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pHi regulation in Ehrlich mouse ascites tumor cells: Role of sodium-dependent and sodium-independent chloride-bicarbonate exchange
AbstractpHi recovery in acid-loaded Ehrlich ascites tumor cells and pHi maintenance at steady-state were studied using the fluorescent probe BCECF.Both in nominally HCO3−-free media and at 25 mmExpand
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Taurine transport systems in the ciliate protozoanTetrahymena pyriformis
SummaryTetrahymena pyriformis cultivated in the presence of 1 mM taurine prior to transfer of the cells to non-nutrient medium express an enhanced capacity for concentrative taurine uptake and forExpand
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A relationship between amino acid and sodium transport in Tetrahymena pyriformis.
Abstract 1. 1. In populations of Tetrahymena pyriformis, synchronized for cell divisions by heat treatment, the inhibitory action of p- dl -fluorophenylalanine and l -lysine on the first synchronousExpand
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Activation of Cl-dependent K transport in Ehrlich ascites tumor cells.
N-ethylmaleimide (NEM) treatment of steady-state Ehrlich cells induces a substantial net loss of cellular KCl and cell shrinkage. The majority of the initial K loss is Cl dependent. From estimates ofExpand
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pH Regulation in Sensitive and Multidrug Resistant Ehrlich Ascites Tumor Cells
Maintenance and regulation of intracellular pH (pHi) was studied in wild-type Ehrlich ascites tumor cells (EHR2) and five progressively daunorubicin-resistant, P-glycoprotein (P-gp)-expressingExpand
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