• Publications
  • Influence
Clinical and sociodemographic study of vitiligo.
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Impact of Performance Appraisal and Working Environment on the Job Satisfaction and Attrition Problem in the Indian IT Industry
This new millennium is witnessing intense competition which is profoundly impacting the business environment. The emerging economy, in which services, communications and information technologies playExpand
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Liquid secretion inhibitors reduce mucociliary transport in glandular airways.
Because of its possible importance in cystic fibrosis (CF) pulmonary pathogenesis, the effect of anion and liquid secretion inhibitors on airway mucociliary transport was examined. When excisedExpand
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Regulation of the Epithelial Na+ Channel by the Protein Kinase CK2*S⃞
CK2 is a ubiquitous, pleiotropic, and constitutively active Ser/Thr protein kinase that controls protein expression, cell signaling, and ion channel activity. Phosphorylation sites for CK2 areExpand
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Inhibition of Protein Kinase CK2 Closes the CFTR Cl- Channel, but has no Effect on the Cystic Fibrosis Mutant ΔF508-CFTR
Background: Deletion of phenylalanine-508 (ΔF508) from the first nucleotide-binding domain (NBD1) in the wild-type cystic fibrosis (CF) transmembrane-conductance regulator (wtCFTR) causes CF.Expand
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The formation of the cAMP/protein kinase A-dependent annexin 2-S100A10 complex with cystic fibrosis conductance regulator protein (CFTR) regulates CFTR channel function.
Cystic fibrosis results from mutations in the cystic fibrosis conductance regulator protein (CFTR), a cAMP/protein kinase A (PKA) and ATP-regulated Cl(-) channel. CFTR is increasingly recognized as aExpand
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Making human nasal cilia beat in the cold: a real time assay for cell signalling.
Human nasal epithelium must adapt to cold climates, and yet, in vitro, human ciliary beat frequency (CBF) is zero at 4 degrees C. Similarly, hibernating mammals do not die of pneumonia despite a coreExpand
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Defective formation of PKA/CnA-dependent annexin 2-S100A10/CFTR complex in DeltaF508 cystic fibrosis cells.
Cystic fibrosis (CF) is characterised by impaired epithelial ion transport and is caused by mutations in the cystic fibrosis conductance regulator protein (CFTR), a cAMP/PKA and ATP-regulatedExpand
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Prevention and management of neonatal hypoglycaemia.
  • Anil C. Mehta
  • Medicine
  • Archives of disease in childhood. Fetal and…
  • 1 January 1994
How do normal neonates adapt to extrauterine metabolism? When the umbilical cord is clamped, the neonate faces a metabolic problem: the insulin dominated fetal milieu, so essential for fetal growth,1Expand
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Cystic fibrosis transmembrane regulator fragments with the Phe508 deletion exert a dual allosteric control over the master kinase CK2.
Cystic fibrosis mostly follows a single Phe508 deletion in CFTR (cystic fibrosis transmembrane regulator) (CFTRDeltaF508), thereby causing premature fragmentation of the nascent protein withExpand
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