André Langer

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The formation of “Advanced Glycation End products” (AGEs) is an inevitable consequence of mammalian glucose metabolism. AGE-mediated protein-protein crosslinks lead to detergent-insoluble and protease-resistant protein aggregates, and in Alzheimer’s disease (AD) extra cellular senile plaques (SPs) and intracellular neurofibrillary tangles (NFTs) have been(More)
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