Adriana A Reyes Barcelo

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BACKGROUND Self-assembly of the amyloid-beta peptide (Abeta) has been implicated in the pathogenesis of Alzheimer's disease (AD). As a result, synthetic molecules capable of inhibiting Abeta self-assembly could serve as therapeutic agents and endogenous molecules that modulate Abeta self-assembly may influence disease progression. However, increasing(More)
Background: Self-assembly of the amyloid- peptide (A) has been implicated in the pathogenesis of Alzheimer's disease (AD). As a result, synthetic molecules capable of inhibiting A self-assembly could serve as therapeutic agents and endogenous molecules that modulate A self-assembly may influence disease progression. However, increasing evidence(More)
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