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  • Bo Li, Christiane Chauvin, +8 authors Eric Fontaine
  • Medicine, Biology
  • Biochimica et biophysica acta
  • 2012 (First Publication: 1 September 2012)
  • Inhibition of the mitochondrial permeability transition pore (PTP) has proved to be an effective strategy for preventing oxidative stress-induced cell death, and the pore represents a viable cellularExpand
  • Christelle Villeneuve, Céline Guilbeau-Frugier, +17 authors Jeanne Mialet-Perez
  • Medicine, Biology
  • Antioxidants & redox signaling
  • 2013
  • AIMS Oxidative stress and mitochondrial dysfunction participate together in the development of heart failure (HF). mRNA levels of monoamine oxidase-A (MAO-A), a mitochondrial enzyme that producesExpand
  • Thomas Bochaton, C. Crola-Da-Silva, +8 authors Delphine Baetz
  • Medicine, Biology
  • Journal of molecular and cellular cardiology
  • 2015 (First Publication: 1 July 2015)
  • RATIONALE How ischemic postconditioning can inhibit opening of the mitochondrial permeability transition pore (PTP) and subsequent cardiac myocytes death at reperfusion remains unknown. RecentExpand
  • Abdallah Gharib, C. Balende, +5 authors J. F. Pujol
  • Biology, Medicine
  • Journal of neurochemistry
  • 1999 (First Publication: 18 January 2002)
  • Biochemical approaches were used in freely moving rats to determine, under steady-state conditions, the brain/arterial plasma partition coefficients of L-tryptophan and alpha-[3H]methyl-L-tryptophan,Expand
  • R. Cespugli, Nicole Sarda, Abdallah Gharib, Héléne Faradji, N. Chastrette
  • Psychology, Medicine
  • Experimental Brain Research
  • 1986 (First Publication: 1 November 1986)
  • SummaryDifferential pulse voltammetry was performed in rats chronically implanted with carbon fiber electrodes in the caudate (n.Cd) and raphe dorsalis (n.RD) nuclei. The electrochemical signalExpand
  • Geoffrey Teixeira, Maryline Abrial, +5 authors Abdallah Gharib
  • Biology, Medicine
  • Journal of molecular and cellular cardiology
  • 2013 (First Publication: 1 March 2013)
  • Reperfusion of the heart after an ischemic event leads to the opening of a nonspecific pore in the inner mitochondrial membrane, the mitochondrial permeability transition pore (mPTP). Inhibition ofExpand