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Amnesia induced in mice by centrally administered β-amyloid peptides involves cholinergic dysfunction
Substantial evidences suggest that the increased cerebral deposition, and neurotoxic action of the beta-amyloid peptide, the major constituent of senile plaques, may represent the underlying cause ofExpand
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Axonal plasticity and functional recovery after spinal cord injury in mice deficient in both glial fibrillary acidic protein and vimentin genes
The lack of axonal regeneration in the injured adult mammalian spinal cord leads to permanent functional disabilities. The inability of neurons to regenerate their axon is appreciably due to anExpand
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Activation of Locomotion in Adult Chronic Spinal Rats Is Achieved by Transplantation of Embryonic Raphe Cells Reinnervating a Precise Lumbar Level
Traumatic lesions of the spinal cord yield a loss of supraspinal control of voluntary locomotor activity, although the spinal cord contains the necessary circuitry to generate the basic locomotorExpand
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Modulation by neurosteroids of the in vivo (+)‐[3H]SKF‐10,047 binding to σ1 receptors in the mouse forebrain
Recent reports suggest an interaction between neuro‐(active)steroids and sigma1 (σ1) receptors, affecting biochemical parameters as well as physiological responses mediated by σ1 ligands in theExpand
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Acute exposure to GSM 900-MHz electromagnetic fields induces glial reactivity and biochemical modifications in the rat brain
The worldwide proliferation of mobile phones raises the question of the effects of 900-MHz electromagnetic fields (EMF) on the brain. Using a head-only exposure device in the rat, we showed that aExpand
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Effects of radiofrequency exposure on the GABAergic system in the rat cerebellum: clues from semi-quantitative immunohistochemistry
The widespread use of cellular phones raises the problem of interaction of electromagnetic fields with the central nervous system (CNS). In order to measure these effects on neurotransmitter contentExpand
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In vitro aggregation facilitates β-amyloid peptide-(25–35)-induced amnesia in the rat
Abstract The β-amyloid peptide-(25–35) fragment, but not β-amyloid peptide-(1–28), shares with β-amyloid protein-(1–42) the ability to self-aggregate and to induce neurotoxicity in vitro. This studyExpand
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The modulation by neurosteroids of the scopolamine-induced learning impairment in mice involves an interaction with sigma1 (σ1) receptors
Neurosteroids have been reported to modulate learning and memory processes in aged animals and in pharmacological models of amnesia. We report here the effects of dehydroepiandrosterone sulfateExpand
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Distribution of p120 catenin during rat brain development:: potential role in regulation of cadherin-mediated adhesion and actin cytoskeleton organization
p120 catenin (p120ctn) is implicated in the regulation of cadherin-mediated adhesion and actin cytoskeleton remodeling. The interaction of cytoplasmic p120ctn with the guanine exchange factor Vav2 isExpand
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Autoradiographic mapping of 5-HT1, 5-HT1A, 5-HT1B and 5-HT2 receptors in the rat spinal cord
The distribution of 5-HT1, 5-HT1A, 5-HT1B and 5-HT2 receptors in the rat spinal cord was investigated with quantitative autoradiography. Receptors were labeled respectively with [3H]serotoninExpand
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