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Respiratory burst in human neutrophils.
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Identification and characterization of SarH1, a new global regulator of virulence gene expression in Staphylococcus aureus
TLDR
It is shown that both the agr‐dependent suppression of protein A production and the sarA‐dependent stimulation of alpha‐toxin production is mediated via a new regulator, SarH1, which belongs to a family of Sar homologues.
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Ligand recognition and activation of formyl peptide receptors in neutrophils
TLDR
The current knowledge about structure and function of two closely related neutrophil G proteincoupled chemoattractant receptors is summarized, which was the first characterized member in the FPR family, and the closely related FPRL1, also called LXA4 receptor, as this eicosanoid was thefirst specific agonist described for the receptor.
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Phosphorylation of uridine and cytidine nucleoside analogs by two human uridine-cytidine kinases.
TLDR
The cloning and recombinant expression of the two human UCKs will be important for development of novel pyrimidine ribonucleoside analogs and the characterization of their pharmacological activation.
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Galectin-3 activates the NADPH-oxidase in exudated but not peripheral blood neutrophils.
TLDR
The major conclusion of the study is that galectin-3 is a potent stimulus of the neutrophil respiratory burst, provided that the cells have first experienced an extravasation process.
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Galectin-3 contributes to neonatal hypoxic–ischemic brain injury
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Secretion of heparin-binding protein from human neutrophils is determined by its localization in azurophilic granules and secretory vesicles.
TLDR
The findings suggest that HBP can be synthesized or targeted to easily mobilized compartments at a late stage of neutrophil maturation, and may be important for proinflammatory functions of this protein, such as the alteration of vascular permeability.
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Neutrophil NET formation is regulated from the inside by myeloperoxidase-processed reactive oxygen species.
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Galectins as inflammatory mediators
TLDR
This review describes how the galectins may be involved in several parts of the inflammatory process, including the recruitment of neutrophils into an infected tissue and the recognition and killing of bacteria by activation of the tissue destructive phagocytic respiratory burst.
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Measurement of respiratory burst products generated by professional phagocytes.
TLDR
Three of these techniques, chemiluminescence amplified by luminol/ isoluminol, the absorbance change following reduction of cytochrome c, and the fluorescence increase upon oxidation of p-hydroxyphenylacetate, are described in detail in this chapter.
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