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Lipoprotein-antibody immune complexes. Their catabolism and role in foam cell formation.
Investigations have shown that during 4-h incubation the uptake of [125I]LDL-anti-apo B IgG complex by human lung fibroblasts was lower than uptake of free LDL, whereas mouse peritoneal macrophages took up immune complex more actively than free LDL.
Accumulation of cholesteryl esters in macrophages incubated with human lipoprotein-antibody autoimmune complex.
It is concluded that formation of LP-Ab autoimmune complexes may play an important role in the formation of atherosclerotic lesions of the arteries.
[ABCG1 transporter gene expression in peripheral blood mononuclear cells of patients with atherosclerosis].
Accumulation of cholesterol in arterial wall macrophages is a main hallmark of atherosclerosis. The ABCG1 transporter mediates cholesterol efflux to high density lipoproteins (HDL) and plays an
ABCA1 gene expression in peripheral blood lymphocytes and macrophages in patients with atherosclerosis
It is suggested that the expression level of ABCA1 gene may regulate atherosclerosis development and is found to play an important role in cholesterol transport from peripheral tissues.