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Protein measurement with the Folin phenol reagent.
Protein Measurement with the Folin
Policy effects of variations in pH, time of reaction, and concentration of reactants, permissible levels of reagents commonly used in handling proteins, and interfering substances are studied.
Decreased lung ischemia-reperfusion injury in rats after preoperative administration of cyclosporine and tacrolimus.
- B. Krishnadasan, B. Naidu, M. Mulligan
- Medicine, BiologyThe Journal of thoracic and cardiovascular…
- 1 April 2002
Cyclosporine and tacrolimus treatment before reperfusion was protective against lung ischemia-reperfusion injury in rats, and the decreased expression of cytokine messenger RNA indicates that both cyclosporines and tacrosporine may exert their protective effects at the pretranscriptional level.
Regulation of chemokine expression by cyclosporine A in alveolar macrophages exposed to hypoxia and reoxygenation.
Oxidative Stress Induces NF-κB Nuclear Translocation Without Degradation of IκBα
Investigation of human cardiac tissue obtained during surgical procedures requiring cardiopulmonary bypass revealed prominent NF-κB activation after hearts had been exposed to ischemia and reperfusion.
An essential role for NF-κB in the cardioadaptive response to ischemia
Inhibition of the initial wave of NF‐κB activity in rat muscle reduces ischemia/reperfusion injury
It is demonstrated that NF‐κB is activated during reperfusion in a biphasic manner and that the regulation of the initial phase of NF-κB activation affords physiological protection against a severe ischemic stress.
An essential role for NF-kappaB in the cardioadaptive response to ischemia.
The cytoprotective effects induced by IP require activation of NF-kappaB, and preconditioned myocardium showed enhanced regional contractile function throughout reperfusion.
Oxidative stress induces NF-kappaB nuclear translocation without degradation of IkappaBalpha.
- T. Canty, E. Boyle, A. Farr, E. N. Morgan, E. Verrier, T. Pohlman
- Biology, MedicineCirculation
- 9 November 1999
A stimulus-specific mechanism of NF-kappaB activation in endothelial cells that acts independently of IkappaBalpha degradation and may require tyrosine phosphorylation is demonstrated.