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Nitric oxide in pancreatic secretion and hormone-induced pancreatitis in rats
TLDR
It is concluded that NO is involved in the stimulation of pancreatic secretion in vivo and exhibits a beneficial effect on pancreatitis, probably by improving the pancreatic blood flow. Expand
Effects of Exercise on Appetite and Food Intake Regulation
TLDR
The evidence to date emphasizes the need to increase physical exercise levels, particularly because of the high prevalence of obesity, and the mechanisms behind the post exercise adjustments in short-term appetite control, and their long-term consequences are needed. Expand
Role of epidermal growth factor in healing of chronic gastroduodenal ulcers in rats.
TLDR
It is suggested that cell proliferation is an important factor in healing of gastric and duodenal ulcerations and that EGF plays an important role in ulcer healing due to its mitogenic action. Expand
Role of epidermal growth factor, prostaglandin, and sulfhydryls in stress-induced gastric lesions.
TLDR
The presence of salivary glands attenuates the stress ulcerogenesis probably by releasing epidermal growth factor which acts, in part, by enhancing ornithine decarboxylase activity, mucosal growth, and prostaglandin and glutathione formation. Expand
Stimulation of pancreatic growth by secretin, caerulein, and pentagastrin.
TLDR
Whether secretin also alters pancreatic growth by itself and whether it influences the trophic response to caerulein, a structural and functional homolog of cholecystokinin, is examined. Expand
Transforming growth factor alpha and epidermal growth factor in protection and healing of gastric mucosal injury.
TLDR
Pretreatment with indomethacin decreased in part the protection by TGF and EGF against ethanol-induced damage and virtually abolished the protective action of these peptides against stress-induced injury. Expand
Dual, time-dependent deleterious and protective effect of anandamide on the course of cerulein-induced acute pancreatitis. Role of sensory nerves.
TLDR
The effect of anandamide on the severity of acute pancreatitis depends on the phase of this disease, and Sensory nerves are involved in the mechanism of this biphasic effect ofAnandamide. Expand
Effect of ischemic preconditioning on pancreatic regeneration and pancreatic expression of vascular endothelial growth factor and platelet-derived growth factor-A in ischemia/reperfusion-induced
TLDR
Ischemic preconditioning reduces pancreatic damage and accelerates pancreatic healing in the course of ischemia/reperfusion-induced pancreatitis, and was associated with improvement of pancreatic blood flow. Expand
Modulation of pancreatic enzyme secretion by melatonin and its precursor; L-tryptophan. Role of CCK and afferent nerves.
TLDR
It is concluded that melatonin or its precursor; L-tryptophan stimulates pancreatic enzyme secretion via stimulation of CCK release and activation of duodeno-pancreatic reflexes. Expand
Stimulation of sensory nerves and CGRP attenuate pancreatic damage in ischemia/reperfusion induced pancreatitis.
TLDR
Stimulation of sensory nerves protects the pancreas against damage evoked by I/R, whereas ablation of these nerves aggravates tissue damage in the Pancreas exposed to I/S, partly dependent on CGRP release. Expand
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