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Sertoli cell differentiation is induced both cell-autonomously and through prostaglandin signaling during mammalian sex determination.
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Severe nasal clefting and abnormal embryonic apoptosis in Alx3/Alx4 double mutant mice.
TLDR
A significant increase in apoptosis localised in the outgrowing frontonasal process in embryonic day 10.0 of Alx3/Alx4 double mutant embryos is proposed to be the underlying cause of the subsequent malformations.
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Jaw transformation with gain of symmetry after Dlx5/Dlx6 inactivation: Mirror of the past?
TLDR
This finding supports the notion that Dlx genes are homeotic genes associated with morphological novelty in the vertebrate lineage and first homeotic‐like transformation found in this Hox‐free region after gene inactivation.
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Expression profiling of purified mouse gonadal somatic cells during the critical time window of sex determination reveals novel candidate genes for human sexual dysgenesis syndromes.
TLDR
A novel reporter mouse line is described that expresses enhanced green fluorescent protein under the control of an Sf1 promoter fragment, marking Sertoli and granulosa cell precursors during the critical period of sex determination.
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Genetics of shoulder girdle formation: roles of Tbx15 and aristaless-like genes
TLDR
Studying the impact of Tbx15, Gli3, Alx4 and related genes on formation of the skeletal elements of the mouse shoulder and pelvic girdles revealed aspects of their genetic relationship, as well as functions that had been previously masked due to functional redundancy.
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Vertebrate aristaless-related genes.
TLDR
Aristaless-related genes, a subset of the Paired-related homeobox genes, have in the past few years emerged as a group of regulators of essential events during vertebrate embryogenesis, and have been shown to have many different roles, including functions in the pituitary, left-right determination and limb development.
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Expression patterns of group-I aristaless-related genes during craniofacial and limb development
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A high-resolution anatomical ontology of the developing murine genitourinary tract.
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Mouse model of split hand/foot malformation type I
TLDR
This is the first evidence that the role of dll/Dlx genes in appendage development is conserved from insects to mammals and proves their involvement in split hand/foot malformation type I SHFM1.
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Msx1 and Dlx5 act independently in development of craniofacial skeleton, but converge on the regulation of Bmp signaling in palate formation
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