A H Hadj Aissa

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BACKGROUND & AIMS Renal impairment is a documented complication in long-term parenteral nutrition (LTPN) patients. However, the aetiologies have remained elusive. The aim of this study was to evaluate the impact of parenteral nutrition, digestive status, and hydration level on renal function in LTPN patients. METHODS In a prospective study of 40 LTPN(More)
In order to assess the main characteristics of the prostaglandin (PG) biosynthesis by the isolated perfused rat kidney, the urinary and venous outputs of PGE2, PGF2alpha, 6-keto-PGF1alpha and of thromboxane (Tx)B2 were followed during 120 min after an equilibration period of 30 min. Single pass kidneys were perfused with a Krebs-Henseleit solution added(More)
We describe three methods for extraction of main tissular cations : a) dry ashing method; b) wet ashing method with HNO3; c) wet method on fresh tissue with HCIO4. This study allows the determination of tissular concentrations of calcium, magnesium in renal cortex of the Rat. Our findings show : a loss of Na, K cations probably by volatilization with dry(More)
1 The determination of Na, Ca, Mg, and K concentrations was performed in four different regions of the dog kidney (cortex, outer medulla, inner medulla, and papilla) during antidiuresis and during an osmotic diuresis. 2 The results show a medullary concentration gradient for calcium. This gradient is much higher than that found for sodium. 3 An inverse(More)
An increased urinary excretion of thromboxane (Tx)B2 (Geoffroy & al., Hypertension 1986, 4 suppl 3: S37) and an elevated renal sympathetic activity (Sautel & al., Am J Physiol 1988, 255: H736) were simultaneously observed in the developing genetically hypertensive rat of the Lyon strain (LH). In the present work, the relationship between the adrenergic(More)
Four beta-blockers: atenolol 15 mg i.v., metoprolol 25 mg i.v., acebutolol 45 mg i.v. and labetalol 50 mg i.v. were administered to 106 patients suffering from hypertension and with normal or disturbed renal function. During an identical test protocol, measurements of clearance of inulin, of PAH, urine sodium and the fraction of sodium excreted were(More)
In 11 patients with advanced renal failure, chronic treatment with ranitidine decreased plasma immunoreactive parathormone (PTH) without affecting phosphate reabsorption or urinary excretion of cyclic AMP. No significant changes in glomerular filtration rate and in urinary excretion of electrolytes were evident.