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We measured lobar hypoxic pulmonary vasoconstriction (HPV) caused by both absorption atelectasis (AA) and nitrogen ventilation (N2) during conditions of a) open chest and positive-pressure ventilation (PPV), b) closed chest ad PPV, and c) closed chest and spontaneous ventilation (SV) and compared conditions a with b and b with c. In eight(More)
We tested the hypothesis that decreases in alveolar O2 pressure (PAO2) of a large lung compartment would, through decreases in arterial O2 pressure (PaO2) and mixed venous O2 pressure (PVO2), result in decreases in PAO2 of the remaining small lung compartment; thus large-compartment hypoxic pulmonary vasoconstriction (HPV) would be accompanied by(More)
Based on previous whole lung findings, the authors tested the hypothesis that lobar hypoxic pulmonary vasoconstriction (HPV) would be potentiated by repeated intermittent lobar hypoxic challenges. In sixteen open-chested pentobarbital-anesthetized dogs they found that repetitive hypoxia of the left lower lobe (LLL) (Group I = LLL nitrogen ventilation, n =(More)
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